陈兴祥
博士生导师
所在单位：动物医学院
教师姓名：陈兴祥
学历：博士研究生毕业
办公地点：教学动物医院
学位：农学博士学位
职称：教授
毕业院校：南京农业大学
学科：临床兽医学
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科研论文
- 甘芳,周亚娇,后丽丽,钱刚,陈兴祥,黄克和.Ochratoxin A induces nephrotoxicity and immunotoxicity through different MAPK signaling pathways in PK15 cells and porcine primary splenocytes,CHEMOSPHERE,2017,182(0):630-637(参与作者)
- 甘芳,后丽丽,周亚娇,刘云欢,黄达,陈兴祥,黄克和.Effects of ochratoxin A on ER stress, MAPK signaling pathway and autophagy of kidney and spleen in pigs,ENVIRONMENTAL TOXICOLOGY,2017,32(10):2277-2286(参与作者)
- 钱刚,刘丹丹,胡俊发,甘芳,后丽丽,陈兴祥,黄克和.Ochratoxin A-induced autophagy in vitro and in vivo promotes porcine circovirus type 2 replication,CELL DEATH & DISEASE,2017,8:-(参与作者)
- 王洪,陈颖,翟年惠,陈兴祥,甘芳,胡丽,黄克和.Ochratoxin A-Induced Apoptosis of IPEC-J2 Cells through ROS-Mediated Mitochondrial Permeability Transition Pore Opening Pathway,JOURNAL OF AGRICULTURAL AND FOOD CHEMISTRY,2017,65(48):10630-10637(参与作者)
- 薛红霞,甘芳,钱刚,胡俊发,郝澍,徐静,陈兴祥,黄克和.Astragalus polysaccharides attenuate PCV2 infection by inhibiting endoplasmic reticulum stress in vivo and in vitro,SCIENTIFIC REPORTS,2017,7(0):-(参与作者)
- 张哲纤,甘芳,薛红霞,刘云欢,黄达,Khan, Alam Zeb,陈兴祥,黄克和.Nephropathy and hepatopathy in weaned piglets provoked by natural ochratoxin A and involved mechanisms,EXPERIMENTAL AND TOXICOLOGIC PATHOLOGY,2016,68(4):205-213(通讯作者)
- 甘芳,胡志华,黄钰,薛红霞,黄达,钱刚,胡俊发,陈兴祥,王恬,黄克和.Overexpression of pig selenoprotein S blocks OTA-induced promotion of PCV2 replication by inhibiting oxidative stress and p38 phosphorylation in PK15 cells,ONCOTARGET,2016,7(15):20469-20485(参与作者)
- 黄克和,刘瑾,陈兴祥,廖顺发,黄达,叶耿坪.Feeding glycerol-enriched yeast culture improves lactation performance, energy status, and hepatic gluconeogenic enzyme expression of dairy cows during the transition period,JOURNAL OF ANIMAL SCIENCE,2016,94(6):2441-2450(参与作者)
- 庄腾寒,徐海滨,郝澍,任飞,陈兴祥,潘翠玲,黄克和.Effects of selenium on proliferation, interleukin-2 production and selenoprotein mRNA expression of normal and dexamethasone-treated porcine splenocytes,Research in Veterinary Science,2015,98(2):59-65(参与作者)
- 陈兴祥,石秀丽,甘芳,黄达,黄克和.Glutamine starvation enhances PCV2 replication via the phosphorylation of p38 MAPK, as promoted by reducing glutathione levels,VETERINARY RESEARCH,2015,46:-(第一作者)
